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The Cytokine Reference is the most complete online work for anyone interested in the role of cytokines in host defense processes.

With essential and comprehensive information on all known cytokines, including chemokines, growth factors and neuropeptides, it includes hundreds of entries with detailed descriptions of cytokine genes, proteins, cell sources, activities, receptors, receptor signal transduction, gene activation, pathophysiology, therapeutics and techniques. This encyclopedic reference is compiled by a panel of global experts and will include extensive tables and diagrams. In addition to being a regularly updated and searchable database, the Cytokine Reference includes a two-volume print set.

Cytokines are produced by virtually all the cells in our bodies, and regulate the systems that defend us against infection, tumors and trauma. Cytokines act in a complex and inter-related manner that is becoming increasingly difficult to decipher. This database and publication enables all those working in the field to find the information they need about this ever expanding field.

Key Features

• On-line version includes full color diagrams and sequences, as well as links to on-line databases, including MEDLINE, PDB, SWISSPRO, and GenBank
• On-line extensively linked database is highly functional and allows you to reach the data in a variety of ways, by cytokine, by cell type, or by disease
• On-line version offers regularly up-dated entries and addition of new entries as research uncovers further information
• On-line database allows extensive links at chapter sub-heading level, guiding you through the maze of inter-relationships
• Print version has 2260 pages of detailed and highly structured information on all the cytokines and other mediators of host defense
• Over 190 chapters contributed by international experts and edited by 6 world-renowned scientists
• Every cytokine or receptor chapter uses the same highly structured sub-headings, ensuring that the information is complete, comprehensive, and easy to find.